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Sakai Lab
 BIOGRAPHICAL SKETCH
Name: Eric Carlson
Role on Project: Postdoctoral Fellow
Institution & Location
Degree
Year(s) Field of
Study
University of Minnesota B.S. 1997
Biology
University of
North Dakota Ph.D. 1997-1998 Pharmicology/Toxicology
Oregon Health & Science University Ph.D. 2006
Cell & Developmental Biology
Major Research Interest:
Extracellular matrix proteins as biomarkers
for disease pathogenesis.
Research and/or Professional
Experience:
2006-present
Postdoc with Dr. Lynn
Sakai, Shriners Hospitals for Children, Portland, Oregon
1999-2006
Graduate student with Dr. William R. Skach, Department of Cell and
Developmental Biology, Oregon Health & Science University, Portland,
Oregon
1997-1998
Graduate Student with Dr. James Drewett, Department of Pharmacology
and Toxicology, University of North Dakota, Grand Forks, North
Dakota
Peer-Reviewed Publications:
Shibatani,
T., Carlson, E.J., Larabee, F., McCormack, A.L.,
Fruh, K., Skach, W.R.
Global organization and function of mammalian cytosolic proteasome
pools: Implications for PA28 and 19S regulatory complexes.
Mol Biol Cell, 17:4962-71. 2006.
Carlson,
E.J., Pitonzo, D., Skach, W.R.
p97 functions as a nonessential auxiliary factor for TM
domain extraction during CFTR ER-associated degradation. EMBO J, 25:
4557-66. 2006.
Oberdorf, J. Carlson, E.J., Skach, W.R.
Uncoupling proteasome peptidase and ATPase activities results
in cytosolic release of an ER polytopic protein.
J Cell Sci, 119: 303-13.
2006.
Carlson, E., Bays, N.
David, L., Skach, W.R.
Reticulocyte lysate as a model system to study endoplasmic reticulum
membrane protein degradation.
Methods Mol Biol, 301: 185-205.
2005.
Oberdorf, J., Carlson, E.J., Skach, W.R.
Redundancy of mammalian proteasome b-subunit
function during endoplasmic reticulum associated degradation.
Biochem, 40: 13397-405. 2001.
Roy,
C.N., Carlson, E.J., Anderson,
E.L., Basava, A., Starnes, S.M., Feder, J.N.,
Enns,
C.A.
Interactions of the ectodomain of HFE with the transferrin
receptor are critical for iron homeostasis in cells.
FEBS Letters, 484: 271-74. 2000.
Kreklau, E.L., Carlson, E.J.,
Drewett, J.G. Nitric
oxide inhibits human aldosteronogenesis without guanylate cyclase
stimulation. Mol and
Cell Endocrin, 153: 103-11. 1999.
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